Vaccine Induced inflammation Linked To Epidemic Of type 2 diabetes And Metabolic Syndrome
Newly published data by Dr. J. Barthelow Classen in The Open Endocrinology Journal shows a 50% reduction of type 2 diabetes occurred in Japanese children following the discontinuation of a single vaccine, a vaccine to prevent tuberculosis. This decline occurred at a time when there is a global epidemic of type 2 diabetes and metabolic syndrome, which includes obesity, altered blood cholesterol levels, high blood pressure, and increased blood glucose resulting from insulin resistance.
Classen proposes a new explanation for the epidemic of both insulin dependent diabetes (type 1 diabetes), which has previously been shown to be caused by vaccines and non insulin dependent diabetes (type 2 diabetes). Upon receipt of vaccines or other strong immune stimulants some individuals develop a hyperactive immune system leading to autoimmune destruction of insulin secreting cells. Other individuals produce increased cortisol, an immune suppressing hormone, to suppress the vaccine induced inflammation. The increased cortisol leads to type 2 diabetes and metabolic syndrome. Japanese children have increased cortisol secretion following immunization compared to White children and this explains why Japanese have a relative high rate of type 2 diabetes but low rate of insulin dependent diabetes compared to Whites. The lower cortisol response attributed to type 1 diabetes and the higher cortisol response attributed to type 2 diabetes explains why type 1 diabetics are generally leaner than type 2 diabetics since elevated cortisol causes weight gain.
"The current data shows that vaccines are much more dangerous than the public is lead to believe and adequate testing has never been performed even in healthy subjects to indicate that there is an overall improvement in health from immunization. The current practice of vaccinating diabetics as well as their close family members is a very risky practice," says Dr. J. Barthelow Classen.
Download the published research paper here.